5 edition of The Entero-insular axis found in the catalog.
Head of title: Satellite Symposium to the 10th IDF Meeting, Göttingen, September 7-8, 1979.
|Statement||volume editor W. Creutzfeldt.|
|Series||Frontiers of hormone research -- vol.7|
|Contributions||Creutzfeldt, C., International Diabetes Federation.|
J Miyawaki K, et al., Glucose intolerance caused by a defect in the entero-insular axis: a study in gastric inhibitory polypeptide receptor knockout mice. Proc Natl Acad Sci U S A. Dec 21;96(26) Roux-en-Y gastric bypass (RYGB) surgery produces a significant improvement in glucose metabolism prior to substantial weight loss; this is proposed to result from an enhanced incretin effect secondary to bypass of the duodenum and proximal jejunum. However, the caloric restriction that occurs early after surgery also has beneficial metabolic by: This suggests that improvements in glucose metabolism and insulin resistance following bariatric surgery result in the short term from decreased stimulation of the entero-insular axis .
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Part of the NATO ASI Series book series (volume 80) Abstract Although circulating glucose is the prime regulator of insulin secretion, it is unlikely that the insulin rise occurring in response to a meal is mediated solely by a change in the glucose concentration bathing the B : Susan M.
Wood. The entero-insular axis: implications for The Entero-insular axis book metabolism. Ranganath LR(1).
Author information: Incretins such as glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide 1 (GLP-1) are intestinal hormones that are released The Entero-insular axis book response to ingestion of nutrients, especially by: 46 Ranganath: The entero-insular axis: implications for human metabolism Article in press - uncorrected proof Table 1 The Entero-insular axis book of glucose-dependent insulinotropic polypeptide (GIP) Author: Lakshminarayan Ranganath.
Thieme E-Books & E-Journals The entero-insular axis in type 2 diabetes - incretins as therapeutic agents W. Creutzfeldt. Enteroinsular axis - incretin effect - GIP - GLP-1  amide - GLP-1 for treatment of diabetes mellitus - Dipeptidylpeptidase IV by: Entero-insular axis, as a functionally isolated system including both pancreatic and intestinal hormones, plays an important role in generating satiety signal.
It can be speculated that any disturbance appearing within entero-insular axis may thus Cited by: Entero-insular axis plays an important role in generating satiety signal. Thus disturbances in this axis may influence the course of anorexia nervosa.
The aim of the study was analysis of the function of the hormonal part of the entero-insular axis in girls with anorexia nervosa. The entero-insular axis: implications for human metabolism; Functional polymorphisms in the promoter of the matrix metalloproteinase-9 gene are not linked with significant plasma MMP-9 variations in healthy subjects; Genetic profiling in healthy subjects The Entero-insular axis book the Stanislas cohort based on 24 polymorphisms: effects on biological variablesCited by: Mice with a targeted mutation of the gastric inhibitory polypeptide (GIP) receptor gene (GIPR) were generated to determine the role of GIP as a mediator of signals from the gut to pancreatic β cells.
GIPR−/− mice have higher blood glucose levels with impaired initial insulin response after oral glucose by: The ageing entero-insular axis Article (PDF Available) in Diabetologia 41(11) December with The Entero-insular axis book Reads How we measure 'reads'.
This book reviews advances concerning stimulus-secretion coupling in the pancreatic B-cell and the role of the entero-insular axis, neural pathways and islet peptides in insulin secretion.
The actions of antidiabetic sulphonylurea The Entero-insular axis book and the mechanisms of defective insulin secretion and pancreatic B-cell destruction The Entero-insular axis book also discussed. The book caters for medical students. The former is a restrictive technique, which causes a reduction in the alimentary flow.
The latter induces a prevalent lipid malabsorption. Most Authors agree that surgery modifies the The Entero-insular axis book milieu, acting at several sites, which include the entero-insular axis, the Author: Melania Manco, Laura Leccesi, Geltrude Mingrone.
African plant foods rich in non-starch polysaccharides reduce postprandial blood glucose and insulin concentrations in healthy human subjects - Volume 80 Issue 5 - Uchenna A. Onyechi, Patricia A. Judd, Peter R. EllisCited by: The effect of a mucoadhesive material, prosopis gum, on the entero-insular axis is presented.
Prosopis gum helps in the serum glucose lowering capacity of metformin. The materia. is compatible with prosopis gum and offers a potential as a potential fixed-ratio combination Author: M.U.
Adikwu, A.A. Attama, O. Okorie. This manuscript talks about the study of the effect of oral nutritional supplements with sucromalt and isomaltulose versus standard formula on glycaemic index, entero-insular axis peptides and subjective appetite in patients with type by: 1.
Mechanisms of Gastrointestinal Motility and Secretion It seems that you're in USA. We have a dedicated site Mechanisms of Gastrointestinal Motility and Secretion.
Editors: Bennett, A. (Ed.) Free Preview. Buy this book eBook 71 The Entero-Insular Axis. Thieme E-Books & E-Journals. Horm Metab Res ; 32(10): DOI: /sCited by: Abstract: In this study, bilosome as a drug delivery system for insulin was studied.
Two bilosomal preparations were formulated and assessed. One was prepared using a lipid extract from Soya beans seed (SBE), Palmitic acid (PA) and cholesterol (CH) in the ratio of w/w designated as BI while the other (BII) contained PA and CH as the lipid entity at the ratio of w/w.
Some of these effects are mediated by the release of insulinotropic hormones from the gut as part of the ‘entero-insular axis’.
Alternatively peptides within the islets of Langerhans may act locally by paracrine or neurocrine mechanisms, or through systemic release to perturb insulin release. Get this from a library.
The Entero-insular axis: satellite symposium to the 10th IDF-Meeting, Göttingen, September[W Creutzfeldt; International Diabetes Federation. Congress;]. Oral diabetes-specific nutritional supplements (ONS-D) induce favourable postprandial responses in subjects with type 2 diabetes (DM2), but they have not been correlated yet with incretin release and subjective appetite (SA).
This randomised, double-blind, cross-over study compared postprandial effects of ONS-D with isomaltulose and sucromalt versus standard formula (ET) Cited by: 1. Abstract: In this study, bilosome as a drug delivery system for insulin was studied. Two bilosomal preparations were formulated and assessed.
One was prepared using a lipid extract from Soya beans seed (SBE), Palmitic acid (PA) and cholesterol (CH) in the ratio of w/w designated as BI while the other (BII) contained PA and CH as the lipid entity at the ratio of w/ by: 4.
Effect Of Catch-Up Growth After Food Restriction On The Entero-Insular Axis In Rats. PubMed ID Ding H, Zhu T, Yin X, Liu J, Zhang L, Bernier M, Zhao R. Acta Biochim Biophys Sin (Shanghai).
Jul;46(7)%(1). The general design of this figure is a modification of the entero-insular axis depicted by Creutzfeldt. Overall, the elegant studies by Gasberg and colleagues (3, 4) have demonstrated that the incretin hormones GIP and GLP-1 are rather unequal with respect to their physiological contribution to the incretin effect, thus behaving more like Cited by: 3.
In addition areas of more direct clinical interest such as the entero-insular axis, insulin secretion in diabetes and insulinoma, mechanisms of sulphonylurea action are also covered.
Although appearing in a series entitled Portland Press Research Monographs this book will be appreciated by both the curious scientist/physician and the researcher. Book Author(s): David Levy MD, FRCP.
Consultant Physician Honorary Senior Lecturer. Gillian Hanson Centre, Whipps Cross University Hospital, UK. Queen Mary University of London, London, UK. Search for more papers by this author. First published: 18 February Background: Catch-up growth after food restriction (CUGFR) is characterized by a significant change in food intake which could theoretically lead to the change in glucagon-like peptide-1 (GLP-1) secretion that consequently results in altered functions of pancreatic islets.
Methods: Experimental rats were divided into two groups. Rats in CUGFR group were put on food Cited by: The effects of dietary energy source and tryptophan on hormones of the entero-insular axis and glucose in the early weaned pig after an intragastric infusion of glucose.
Proceedings of the Nutrition Soci A. Rainbird, A. Studies on the physiological action of dietary fibre in the stomach and small intestine of growing by: The mechanisms for these improvements are not completely understood, but hypothesized to be due to a combination of post-operative reduction of caloric intake and changes in the entero-insular axis or incretin : Whitney S Goldner.
She is particularly interested in the role that alterations of the entero-insular axis may play in the pathophysiology of these disorders. Description of Clinical Expertise Dr. De León specializes in the diagnosis and management of monogenic disorders of insulin regulation resulting in diabetes and hypoglycemia.
Tryptophan (TRP) is shown to play original roles in the physiology of pigs. Dietary TRP deficiency induces depression of the appetite leading to reduced growth Cited by: Entero-insular axis. Arch Intern Med –Crossref PubMed Google Scholar; Westerfield M.
The Zebrafish Book. A Guide For The Laboratory Use of Zebrafish (Danio rerio). Eugene, OR: University of Oregon Press, Google Scholar; Wolfe MM, McGuigan JE. Release of gastric inhibitory peptide following a peptone meal in Cited by: Lifestyle Modification Is the First Line Treatment for Type 2 Diabetes.
By Kazuko Masuo. in the short-term from decreased stimulation of the entero-insular axis by restricted calorie intake and in the long-term by decreased release of adipocytokines due to reduced fat mass.
Help us write another book on this subject and reach those : Kazuko Masuo. It has been postulated that gastric bypass improves diabetes through influences on the entero-insular axis, through a humoral effect. Our understanding of the changes in insulin resistance following surgery has been assisted by the documentation of marked falls in plasma insulin levels within days of gastric bypass.
The significance of over- or underproduction of glucagon-like peptide 1 (GLP-1) (7–36) amide in diabetes remains controversial (1,2).
Although it has been proposed that at least one factor contributing to the pathogenesis of type 2 diabetes is desensitization of the GLP-1 receptor on β-cells (2), exogenous administration of GLP-1 in patients with type 2 diabetes Cited by: Background: Well-controlled studies have demonstrated that inpatient hyperglycemia is an indicator of poor clinical outcomes, but the use of diabetes-specific enteral formulas in hospitalized patients remains a topic of great debate.
Methods: In two different protocols, postprandial glycemia and insulinemia were measured in 22 subjects with diabetes fed a Cited by: Adrenal Gland, Vascular System and Hypertension. Adrenal Gland, Vascular System and Hypertension.
Connell, John M. Blackwell Science Ltd value of workshops is the immediacy and informality which allows interpersonal exchange of views to occur. This, sadly, cannot be reï¬ ected two years after the event. Glucose intolerance caused by a defect in the entero-insular axis: A study in gastric inhibitory polypeptide receptor knockout mice Kazumasa Miyawaki, Yuichiro Yamada, Hideki Yano, Hitoshi Niwa, Nobuhiro Ban, Yu Ihara, Akira Kubota, Shinpei Fujimoto, Mariko Kajikawa, Akira Kuroe, Kinsuke Tsuda, Hiroyuki Hashimoto, Tokuyuki Yamashita.
The incretin activity seen in health is due to glucagon-like peptide-1(GLP-1) and gastric inhibitory polypeptide (GIP) (Table 1) (). The incretin axis in the gut-the entero-insular axis: Both the incretins are secreted from two distinct cell types of intestine.
GIP: Secreted from intestinal k-cells most abundant in duodenum and jejunum. Click on the title to browse this issue. Dr. Hirofumi Hashimoto is an internist in Santa Rosa, California pdf is affiliated with one hospital. Glucose intolerance caused by a defect in the entero-insular axis: a study in gastric.She is particularly interested in the role that alterations of the entero-insular axis may play in the pathophysiology of these disorders.
Description of Itmat Expertise Dr. De León’s translational research program focuses on examining the role of the insulinotropic hormone glucagon-like peptide-1 in the pathophysiology of congenital and.Gastric inhibitory ebook receptor.
Gene. Gipr. Organism. Mus musculus (Mouse) Status. Reviewed-Annotation score: Experimental evidence at transcript level i. Function i. This is a receptor for GIP. The activity of this receptor is mediated by G proteins which activate adenylyl cyclase (By similarity).